Expresión del receptor del factor de necrosis tumoral en monocitos infectados por citomegalovirus humano

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Luz María Aguilar Anguiano
José Dártagnan Villalba Magdaleno
Francisco Vianney Ortiz Navarrete
Rosana Pelayo Camacho
Horacio Astudillo de la Vega
Ramón Arturo Valdés Espinosa

Resumen

El citomegalovirus humano (HCMV) es el miembro más representativo de los herpesvirus beta, causa infecciones persistentes y latentes con periodos frecuentes de reactivación. Afecta a todos aquellos que se encuentran inmunosuprimidos e inmunocomprometidos, aumentando su morbilidad y mortalidad.

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1.
Expresión del receptor del factor de necrosis tumoral en monocitos infectados por citomegalovirus humano. RSM [Internet]. 2025 Feb. 18 [cited 2026 Feb. 4];61(6). Available from: https://www.revistasanidadmilitar.org/index.php/rsm/article/view/2265

Referencias

Trine H, Mogensen and Soren R Paludan. Molecular Pathway in Virus-Induced Cytokine Production Microbiology and Molecular Biology Reviews 2001; 65 (1): 131-50.

Chee MS, Bankier AT, et al. Analysis of the protein-codong content of the sequence of human cytomegalovirus AD169. Curr Top Microb Immuno 1990; 1154: 125.

Nordoy I, Muller F, Knut PN, Rollai H, Lien E, Aukrust P. The Role of the Tumor Necrosis Factor System and Interleukin-10 during Cytomegalovirus Infection in Renal Transplant Recipients. The Journal of Infectious Diseases 2000; 181: 51-7.

Grundy JE, Lawson KM, MacCormac LP, Flecher KM. CMVinfected endothelial cells recruit neutrophils by the secretion of C-X-C cytokines and transmit virus by direct neutrophil-endothelial cell contact and during neutrophil transendotelial migration. J Infect Dis 1998; 177: 1465-74.

Yorken JA, Record M, Preval C, Davrinche C, Davignon JL. Distinct Pathways for Tumor Necrosis Alpha and Ceramides in Human Cytomegalovirus Infection. Journal of Virology 1998; 72: 2316-23.

Landini MP, Lazzarotto T, Xu J. Immune Response to CMV Latency-Associated ORF's. Biology of blood and Marrow Transplantation 2001;

(2): 100-8.

Braunwald FW, Wilson I, Kasper M, Longo H. Principios de Medicina Interna, Harrison, 14a Edición, Madrid: Mc Graw Hill, 1998; 2:

-4.

Cotsman D, Mullberg J, Claire L, Chin W et al. ULBPs, Novel MHC class 1-Related Molecules, Bind to CMV Glycoprotein UL16 and Stimulate NK Cytotoxicity through the NKG2D Receptor. Immunity 2001; 14: 123-33.

Van der Berg AP, Meyaard L, Otto SA, Klompmaker U, Mesander G. Cytomegalovirus infection associated with a decreased proliferative capacity and increased rate of apoptosis of peripheral blood lymphocytes. Traspl Proc 1995; 27: 936-8.

Taylor JW, Sissons JGP, Borysiewics LK, Sinclair JH. Monocytes are site of persistence of human cytomegalovirus in peripheral blood mononuclear cells. The Journal of General Virology 1991; 72: 2059-64.

Hahn C, Jores R, Mocarski E. Cytomegalovirus remains latent in a common precursor of dendritic and myeloid cells. The National Academy of Science 1998; 95: 3937-42.

Dankner WM, McCutchan JA, Richman DD, Hirata K, Spector SA. Localization of Human Cytomegalovirus in Peripheral Blood Leukocytes by In Situ Hibridization. The Journal of Infectious Diseases 1990; 161: 31-6.

Sódeberg CN, Fish KN, Nelson JA. Interferon-gama and tumor Necrosis factor-alfa Specifically Induce formation of Cytomegalovirus-permissive Monocytes-derived Macrophages that are refractory to the Antiviral Activity of these Citokines. J Clin Invest 1997; 100 (12):

-63.

Michelsson S, Aclami J, Danelipur D, Bachelerie F, Picard L. Human Cytomegalovirus infection induces transcription and secretion of transforming growth factor B1. J Virol 1994; 68: 5730-7.

Koszinowsky UH, Del Val M, Reddhase MJ. Immune response to cytomegalovirus infection. Curr Top Microbiol Immunol 1990; 154: 189-220.

Hestdal K, Aukrust P, Muller F, Froland S. Dysregulation of Membrane-Bound Tumor Necrosis Factor-? and tumor necrosis factor Receptors on Mononuclear cell in human immunodeficiency Virus Type 1 Infection; Low Percentage of p75-Tumor Necrosis factor Receptor Positive Cell in Patients With Advanced Disease and High Viral Load. Blood 1997; 90 (7): 2670-9.19.

Peschon J, Ruby J, Bluethmann H. Antiviral Activity of Tumor Necrosis Factor (TNF) Is Mediated via p55 abd p75 TNF Receptors. J Exp Med 1997; 186 (9): 1591-6.

Camelo, Lafage M, Galelli A, Lafon M. Selective role for the p55 Kd TNF-? receptor in immune unresponsiveness induced by an acute viral encephalitis. Journal of neuroimmunology 2001; 113: 95-108.

Spiller OB, Morgan BP, Tufaro F, Devine DV. Altered expression of host-encoded complement regulators on human cytomegalovirus-infected cells. Eur J Immunol 1996; 26: 1532-8.

Borysiewickz LK, Sissons GP. Cytotoxic T cells and human herpes virus infections. Curr Top Microbiol Immunol 1994; 189: 123-150.

Yurochko AD, Huand E. Human Cytomegalovirus Binding to Human Monocytes Induced Immunoregulatory Gene Expression. The Journal of Immunology 1999; 162: 4806-16.

Humar A, Louis St, Mazzulli, McGeer A, Lipton J, Messnes H, MacDonald S. Elevated serum Cytokines are associated with Cytomegalovirus Infection and Disease in Bone marrow Transplant recipients. The Journal of Infectious disease 1999; 179: 484-8.

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